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The inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse.
Proceedings of the National Academy of Sciences of the United States of America
, Effects of a synthetic PEG-ylated Tie-2 agonist peptide on endotoxemic lung injury and mortality.
American journal of physiology. Lung cellular and molecular physiology
, New strategies for echocardiographic evaluation of left ventricular function in a mouse model of long-term myocardial infarction.
PloS one
, Angiotensin II Type 2 Receptor Stimulation: A Novel Option of Therapeutic Interference With the Renin-Angiotensin System in Myocardial Infarction?
Circulation
, Guided cardiopoiesis enhances therapeutic benefit of bone marrow human mesenchymal stem cells in chronic myocardial infarction.
Journal of the American College of Cardiology
, GS-6201, a selective blocker of the A2B adenosine receptor, attenuates cardiac remodeling after acute myocardial infarction in the mouse.
The Journal of pharmacology and experimental therapeutics
, Repair of Acute Myocardial Infarction by Human Stemness Factors Induced Pluripotent Stem Cells
Circulation
, Caveolin-1 deficiency exacerbates cardiac dysfunction and reduces survival in mice with myocardial infarction.
American journal of physiology. Heart and circulatory physiology
, Absence of type VI collagen paradoxically improves cardiac function, structure, and remodeling after myocardial infarction.
Circulation research
, IL-10 inhibits inflammation and attenuates left ventricular remodeling after myocardial infarction via activation of STAT3 and suppression of HuR.
Circulation research
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