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Notch1 in bone marrow-derived cells mediates cardiac repair after myocardial infarction.
Circulation
, Oxygen cycling in conjunction with stem cell transplantation induces NOS3 expression leading to attenuation of fibrosis and improved cardiac function
Cardiovascular Research
, Transforming Growth Factor-α Enhances Stem Cell-Mediated Postischemic Myocardial Protection
The Annals of Thoracic Surgery
, Carvedilol enhances mesenchymal stem cell therapy for myocardial infarction via inhibition of caspase-3 expression.
The Journal of pharmacology and experimental therapeutics
, Loss of Apelin exacerbates myocardial infarction adverse remodeling and ischemia-reperfusion injury: therapeutic potential of synthetic Apelin analogues.
Journal of the American Heart Association
, Donor myocardial infarction impairs the therapeutic potential of bone marrow cells by an interleukin-1-mediated inflammatory response.
Science translational medicine
, Angiotensin II Type 2 Receptor Stimulation: A Novel Option of Therapeutic Interference With the Renin-Angiotensin System in Myocardial Infarction?
Circulation
, Guided cardiopoiesis enhances therapeutic benefit of bone marrow human mesenchymal stem cells in chronic myocardial infarction.
Journal of the American College of Cardiology
, GS-6201, a selective blocker of the A2B adenosine receptor, attenuates cardiac remodeling after acute myocardial infarction in the mouse.
The Journal of pharmacology and experimental therapeutics
, Repair of Acute Myocardial Infarction by Human Stemness Factors Induced Pluripotent Stem Cells
Circulation
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