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Carvedilol enhances mesenchymal stem cell therapy for myocardial infarction via inhibition of caspase-3 expression.
The Journal of pharmacology and experimental therapeutics
, Loss of Apelin exacerbates myocardial infarction adverse remodeling and ischemia-reperfusion injury: therapeutic potential of synthetic Apelin analogues.
Journal of the American Heart Association
, Donor myocardial infarction impairs the therapeutic potential of bone marrow cells by an interleukin-1-mediated inflammatory response.
Science translational medicine
, Notch1 in bone marrow-derived cells mediates cardiac repair after myocardial infarction.
Circulation
, Oxygen cycling in conjunction with stem cell transplantation induces NOS3 expression leading to attenuation of fibrosis and improved cardiac function
Cardiovascular Research
, Transforming Growth Factor-α Enhances Stem Cell-Mediated Postischemic Myocardial Protection
The Annals of Thoracic Surgery
, IL-10 inhibits inflammation and attenuates left ventricular remodeling after myocardial infarction via activation of STAT3 and suppression of HuR.
Circulation research
, Preventing cardiac remodeling: the combination of cell-based therapy and cardiac support therapy preserves left ventricular function in rodent model of myocardial ischemia.
The Journal of thoracic and cardiovascular surgery
, Granulocyte colony-stimulating factor treatment plus dipeptidylpeptidase-IV inhibition augments myocardial regeneration in mice expressing cyclin D2 in adult cardiomyocytes.
European heart journal
, Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodeling.
American journal of physiology. Heart and circulatory physiology
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