Publications Library

Carvedilol enhances mesenchymal stem cell therapy for myocardial infarction via inhibition of caspase-3 expression.

Loss of Apelin exacerbates myocardial infarction adverse remodeling and ischemia-reperfusion injury: therapeutic potential of synthetic Apelin analogues.

Donor myocardial infarction impairs the therapeutic potential of bone marrow cells by an interleukin-1-mediated inflammatory response.

Notch1 in bone marrow-derived cells mediates cardiac repair after myocardial infarction.

Oxygen cycling in conjunction with stem cell transplantation induces NOS3 expression leading to attenuation of fibrosis and improved cardiac function

Transforming Growth Factor-α Enhances Stem Cell-Mediated Postischemic Myocardial Protection

Beta3-adrenoreceptor stimulation ameliorates myocardial ischemia-reperfusion injury via endothelial nitric oxide synthase and neuronal nitric oxide synthase activation.

CC chemokine receptor 5 deletion impairs macrophage activation and induces adverse remodeling following myocardial infarction

The inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse.

Effects of a synthetic PEG-ylated Tie-2 agonist peptide on endotoxemic lung injury and mortality.