Publications Library

Ablation of Nkx2-5 at mid-embryonic stage results in premature lethality and cardiac malformation.

Inactivation of Plin4 downregulates Plin5 and reduces cardiac lipid accumulation in mice.

Cardiac-specific deletion of the microtubule-binding protein CENP-F causes dilated cardiomyopathy.

Agonist-independent constitutive activity of angiotensin II receptor promotes cardiac remodeling in mice.

Sustained hemodynamic stress disrupts normal circadian rhythms in calcineurin-dependent signaling and protein phosphorylation in the heart.

Timing of myocardial trpm7 deletion during cardiogenesis variably disrupts adult ventricular function, conduction, and repolarization.

TBX5 drives Scn5a expression to regulate cardiac conduction system function

Cardiomyocyte-specific loss of neurofibromin promotes cardiac hypertrophy and dysfunction.

Cardiac response to pressure overload in 129S1/SvImJ and C57BL/6J mice: temporal- and background-dependent development of concentric left ventricular hypertrophy

Accelerated development of pressure overload-induced cardiac hypertrophy and dysfunction in an RyR2-R176Q knockin mouse model.