Publications Library

Left ventricular diastolic dysfunction in Nrf2 knock out mice is associated with cardiac hypertrophy, decreased expression of SERCA2a, and preserved endothelial function

Myocardial Hypertrophic Remodeling and Impaired Left Ventricular Function in Mice with a Cardiac-Specific Deletion of Janus Kinase 2

Sheng-Mai-San attenuates contractile dysfunction and structural damage induced by chronic intermittent hypoxia in mice

Acute aerobic exercise increases exogenously infused bone marrow cell retention in the heart

Inhibition of MEF2A prevents hyperglycemia-induced extracellular matrix accumulation by blocking Akt and TGF-β1/Smad activation in cardiac fibroblasts

A1 receptor activation attenuates cardiac hypertrophy and fibrosis in response to α1 ­ adrenergic stimulation in vivo . PubMed Commons

CTGF knockout does not affect cardiac hypertrophy and fibrosis formation upon chronic pressure overload

E2F1 suppresses cardiac neovascularization by down-regulating VEGF and PlGF expression.

MicroRNA-221 inhibits autophagy and promotes heart failure by modulating the p27/CDK2/mTOR axis

Aspirin Attenuates Angiotensin II-induced Cardiomyocyte Hypertrophy by Inhibiting the Ca 2+ /Calcineurin-NFAT Signaling Pathway