Publications Library

Original Article Differential Functional Deficits in Type 1 Versus Type 2 Diabetic Mice Models

Maintained right ventricular pressure overload induces ventricular-arterial decoupling in mice

Targeting and modulating infarct macrophages with hemin formulated in designed lipid-based particles improves cardiac remodeling and function

Suppression of NLRP3 Inflammasome Activation Ameliorates Chronic Kidney Disease-Induced Cardiac Fibrosis and Diastolic Dysfunction

Suppression of NLRP3 Inflammasome Activation Ameliorates Chronic Kidney Disease-Induced Cardiac Fibrosis and Diastolic Dysfunction

Elastase-2, an angiotensin II-generating enzyme, contributes to increased Ang II in resistance arteries of mice with myocardial infarction.

UVRAG Deficiency Exacerbates Doxorubicin-Induced Cardiotoxicity

Liposome encapsulated berberine treatment attenuates cardiac dysfunction after myocardial infarction

Bariatric Surgery Ameliorates Diabetic Cardiac Dysfunction by Inhibiting ER Stress in a Diabetic Rat Model

The role of GRIP1 and ephrin B3 in blood pressure control and vascular smooth muscle cell contractility